is a head of the Laboratory of Mitochondrial Biology and Metabolism at the Nencki Institute of Experimental Biology of the Polish Academy of Sciences in Warsaw. From 2022 he is Councilor of European Society for Clinical Investigation (ESCI). In 1995, he graduated from the Faculty of Biology of the University of Warsaw with a Master of Science and next in 1999 completed his PhD at the Nencki Institute of Experimental Biology focusing on the role of mitochondrial permeability transition pore in the uncoupling of the oxidative phosphorylation. In 2002, he joined the prof. R. Rizzuto’s Laboratory at the University of Ferrara where, as a postdoctoral fellow he began studies on the close interactions between endoplasmic reticulum and mitochondria. In 2007, he came back to Poland where in 2016 he was awarded the title of full Professor of Biological Sciences. Prof. Wieckowski is an author / co-author of over 170 publications in refereed journals (as e.g., two papers in Science, two papers in Nature Protocols, two papers in PNAS, Nature Reviews Cardiology, Brain, Biological Reviews, Science Advances, EMBO Reports, two in Cell Reports, Redox Biology and etc.) which were cited more than 12800 times (H-index = 59). His achievements resulted in many prestigious National and European grants and awards. 

Scientific Focus:

Prof. Mariusz R. Wieckowski is leading expert in mitochondrial field, mitochondrial bioenergetics and oxidative stress in the context of several pathological situations. His recent research is focused on different aspects of intracellular oxidative stress (e.g., in the context of mitochondrial disorders) and resolving the issue how does mitochondrial dysfunction affect mitochondrial and cellular function. Moreover, he is investigating the relations between mitochondrial energetic and oxidative stress in tumors as well as pharmacological toxicity to mitochondria. Recently, within the frame of three Horizon 2020 projects he is working on the role of mitochondria and oxidative stress in nonalcoholic fatty liver disease development and progression.